Areas including, cuts and scrapes of the skin, unintentional injections, or areas exposed to the external environment are prone to filovirus entry by virus attachment to cells, endocytosis, and glycoprotein fusion inside the host cell (Martines et al., 2014). The current model for how Ebola and Marburg virus enter host cell explains that through cell receptors, with macrophages and dendritic cells being a main target for early infection, filovirus cellular proteins penetrate tissue obstructions and allow broad tropism for infection causing the infection to have the ability to survive in a variety of circumstantial environments (Martines et al., 2014).