Guillain Barre Syndrome

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Introduction
Guillain-Barré syndrome (GBS) is a complicated degenerative neurological disorder which can be acute or chronic in nature. It is an acquired condition which is characterized by progressive, symmetrical. Proximal and distal tingling and weakness. Acute demyelinating motor and autonomic dysfunction are frequently following a prodromal illness (usually a respiratory or gastrointestinal infection). It is thought to be autoimmune in origin. GBS can cause significant morbidity requiring long hospital in patient stay and significant periods of rehabilitation. Approximately 10–15% of patients require assistance with long term residual disability.

Abstract
This 37 year old previously healthy female school teacher was admitted to James …show more content…
They reported the combination of increased protein concentration with a normal cell count in the CSF, or albumin cytological dissociation, which differentiated the condition from poliomyelitis. Despite the fact that Landry had already reported similar cases in 1859, the combination of these clinical and laboratory features became known as Guillain-Barre syndrome (2).

Guillain-Barré syndrome (GBS) is an acute demyelinating polyneuropathy.Its features are those of ascending motor weakness, sensory and autonomic dysfunction frequently following a prodromal illness (usually a respiratory or gastrointestinal infection). It is thought to be autoimmune in origin. GBS can cause significant morbidity requiring long hospital inpatient stay and significant periods of rehabilitation. Approximately 10–15% of patients require assistance with long term residual disability(5). …show more content…
This patient had the respiratory weakness in the initial phase. Respiratory dysfunction may lead to respiratory failureresulting reduced vital capacity, inspiratoryforce and tidal volume. Further, the atelectasis may develop due to impaired cough, which will lead to arterio-venous shunting and hypoxia,tachypnea and increased work of breathing resulting type 1 respiratory failure. Eventually with increasing weakness may retain CO2 and causes type 2 respiratory failure or complete respiratory arrest. This may be further complicated by infections like in this instance. Patients requiring ventilatory support have less favorableprognosis for neurologicalrecovery,longerhospitalizationsand higher mortality.If diaphragmatic and respiratory muscle weakness has not occurred by 2 weeks into the illness, then assisted ventilation, most probably may not be
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